Photo albums of: June 2012

Saturday, June 9, 2012

Friday, June 8, 2012

What is kawasaki’s disease. Kawasaki (KAH-wah-SAH-ke) disease is a rare childhood disease. It’s a form of a disease called vasculitis (vas-kyu-LI-tis). In Kawasaki disease, the walls of the blood vessels throughout the body become inflamed. The disease can affect any type of blood vessel in the body, including the arteries, veins, and capillaries.
In some cases, Kawasaki disease affects the coronary arteries, which carry oxygen-rich blood to the heart. As a result, a small number of children who have Kawasaki disease may develop serious heart problems. The cause of Kawasaki disease isn’t known. The body’s response to a virus or infection combined with genetic factors may cause the disease. However, no specific virus or infection has been found, and the role of genetics isn’t known.

The disease can’t be passed from one child to another. Your child won’t get it from close contact with a child who has the disease. Also, if your child has the disease, he or she can’t pass it to another child. Kawasaki disease affects children of all races and ages and both genders. It occurs most often in children of Asian and Pacific Island descent. The disease is more likely to affect males, and most cases occur in children younger than 5 years old.
One of the main symptoms of this disease is a fever that lasts longer than 5 days. The fever remains high even after treatment with standard childhood fever medicines. Children who have the disease also may have red eyes, red lips, and redness on the palms of their hands and soles of their feet. These are all signs of inflamed blood vessels. Early treatment is important. It helps reduce the risk that Kawasaki disease will affect the coronary arteries and cause more serious problems.
Kawasaki disease can’t be prevented. However, most children who develop Kawasaki disease fully recover—usually within weeks of getting symptoms. Further problems are rare. In some children, the disease affects the coronary arteries. This can cause serious problems. These children need long-term care and treatment. Researchers continue to look for the cause of Kawasaki disease and better ways to diagnose and treat it. They also hope to learn more about long-term health risks, if any, for people who have had the disease. ( Definition from nhlbi.nih.gov with “What is kawasaki’s disease” )

Venereal disease

Venereal disease. A disease that is contracted and transmitted by sexual contact, caused by microorganisms that survive on the skin or mucus membranes, or that are transmitted via semen, vaginal secretions, or blood during intercourse.
Because the genital areas provide a moist, warm environment that is especially conducive to the proliferation of bacteria, viruses, and yeasts, a great many diseases can be transmitted this way. They include AIDS, chlamydia, genital warts, gonorrhea, syphilis, yeast infections, and some forms of hepatitis.

Sexually transmitted diseases (STDs) are infections that are transmitted during any type of sexual exposure, including intercourse (vaginal or anal), oral sex. a contagious Venereal disease (as gonorrhea or syphilis) that is typically acquired in sexual intercourse. A sexually transmitted disease (STD), also known as sexually transmitted infection (STI) or venereal disease (VD), is an illness that has a significant probability.
Also known as a morbus venereus or sexually transmitted disease (STD).
( From medterms.com about Venereal disease )

Palm diseases

Palm diseases :
Bud Rots of Palm
Fusarium Wilt of Canary Island Date Palm
Fusarium Wilt of Queen Palm and Mexican Fan Palm
Ganoderma Butt Rot of Palms
Graphiola Leaf Spot (False Smut) of Palm
Leaf Spots and Leaf Blights of Palm diseases
Lethal Yellowing (LY) of Palm
Normal “Abnormalities” in Palms
Pestalotiopsis (Pestalotia) Diseases of Palm
Petiole (Rachis) Blight of Palm
Texas Phoenix Palm Decline
Thielaviopsis Trunk Rot of Palm diseases

Palm trees grown in the landscape appear carefree, but they are susceptible to many diseases, insects and nutritional problems. Avoid many of these problems a new batch of problems are out to get your palms, but you can fight back. Insect pests and a number of diseases can attack and damage palms and in extreme cases can kill them. If the growing point of a palm is invaded by insects. Some leaf spot fungi move in as secondary problems on palm leaves that are deficient in nutrients or have received some sort of damage.

The Genetic Epidemiology of Parkinson’s Disease

The Genetic Epidemiology of Parkinson’s Disease. The cause of Parkinson’s disease (PD) is unknown. The major risk factors identified to date are family history, age, and elements of rural living. Nearly one-third of all PD cases are familial, a small subset of which appears autosomal dominant; however, the majority exhibit no clear inheritance pattern. Autosomal dominant PD is genetically heterogeneous: two PD genes have been mapped to chromosomes 2 and 4 and there may be additional as yet unidentified genes.
The common forms of The Genetic Epidemiology of Parkinson’s Disease familial and sporadic cases-appear to involve a complex interplay of genetic susceptibility and environmental exposure. The observations that rural residence and pesticide exposure increase the risk of developing PD, and that a synthetic drug, 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine, can cause parkinsonism, suggest that at least a subset of PD may be caused by a toxin. Furthermore, modest but significant associations have been reported between PD susceptibility and genes that regulate metabolism of drugs and neurotoxins.

There is also evidence for mitochondrial dysfunction in PD, a finding that was recently traced to anomalies in mitochondrial DNA. At the present time, the genetics of PD appear to be complex, involving multiple nuclear genes and possibly mitochondrial genes as well. ( From ncbi.nlm.nih.gov about The Genetic Epidemiology of Parkinson’s Disease ).

Thursday, June 7, 2012

Coronary heart disease symptoms artery

Coronary heart disease symptoms artery. Coronary artery disease is atherosclerosis of the coronary arteries, producing blockages in the vessels which nourish the heart itself.
Atherosclerosis occurs when the arteries become clogged and narrowed, restricting blood flow. Without adequate blood flow from the coronary arteries, the heart becomes starved of oxygen and vital nutrients it needs to work properly.

Coronary heart disease (CHD), also called Coronary heart disease symptoms artery. Without quick treatment, a heart attack can lead to serious problems and even death. If your coronary arteries become partially blocked, it can cause chest pain (angina). In some cases, people may not have symptoms of coronary heart disease. Shortness of breath is most often a symptom of cardiac or pulmonary (lung) disorders. Heart failure and coronary artery disease frequently produce shortness. Treatment for Coronary heart disease symptoms artery depends upon how far the disease has already progressed. Coronary artery disease is the buildup of plaque.
Heart pain, caused by plaque in the arteries, is often one of the first symptoms of coronary artery disease (CAD). Some people who have coronary artery disease and insufficient blood flow to the heart muscle (ischemia) do not have any symptoms. Many people have no symptoms of coronary artery disease before having a heart attack; 63% of women and 48% of men who died suddenly of Coronary heart disease symptoms artery.

Coronary heart disease symptoms artery pictures

Crohn’s disease life expectancy

Crohn’s disease life expectancy. Having Crohn’s Disease doesn’t mean that you are going to drop dead tomorrow, or have a life ten years shorter than everyone else. People with Crohn’s Disease have a relatively normal life expectancy compared to the general population.
You may need to make lifestyle changes to make sure that your Crohn’s disease life expectancy in remission, but on the whole people with Crohn’s Disease can do anything that they put their minds to.

Some lifestyle changes can include diet, getting enough exercise and keeping stress and stress levels in check. By looking after yourself and your body you can enjoy life as much as the next person.
The risk of dying from an acutely serious attack of Crohn’s Disease in this day and age is relatively low. If you had Crohn’s Disease in the 1950’s the risk of death due to a serious attack of Crohn’s was well over 30 percent! ( Detail from healcrohnsnow.com about Crohn’s disease life expectancy )

Crohn’s disease rash

Crohn’s disease rash. Hartnup disease a hereditary disorder of intestinal and renal transport of neutral ?
-amino acids, marked by a pellagra-like skin rash, with transient cerebellar ataxia, constant renal aminoaciduria, and other biochemical abnormalities.
People with Crohn’s disease rash also experience rashes, arthritis, and swollen eyes. The condition is an inflammatory bowel disease. Crohn’s disease can cause symptoms outside the digestive tract, such as joint pain, eye problems, a skin rash, or liver disease. The three most common sites of intestinal involvement in Crohn’s disease are rashes, arthritis, inflammation of the eye, tiredness, and lack of concentration.

Some individuals with Crohn’s disease rash also develop symptoms outside of the gastrointestinal tract; these symptoms include arthritis, skin rash, and inflammation. Crohn’s disease (regional enteritis, granulomatous ileitis, ileocolitis) is a chronic. Some people have reactions such as fever or rash during the infusion. Crohn’s disease rash.

Crohn’s disease symptoms description

Crohn’s disease symptoms description. The commonest Crohn’s disease definition is that it is a form of inflammatory bowel disease that makes the patient’s intestinal tract swell up and in addition, it can cause restricted functioning of that part of the body as well.
In fact, from available Crohn’s disease symptoms description it is also clear that one should make an effort to find out more about what inflammatory bowel diseases are all about so that one is in a better position to understand the true meaning of Crohn’s disease as well as figure out what the consequences of suffering from the disease are.

However, one thing is clear and that is that the medical fraternity has not as yet been able to fathom why a patient’s gastrointestinal tract becomes inflamed though according to findings of researchers, it seems that it is a failure of the immune system that can cause such inflammation and thus lead people to start looking for Crohn’s disease definitions. ( Detail from crohnsdiseasetoday.com on Crohn’s disease symptoms description )

Gaucher disease pathway

Gaucher disease pathway. In relation to Gaucher’s disease, initial results of enzymatic complementation were disappointing, but the reason for this was soon established. Crude enzyme preparations obtained from human tissues such as the placenta were not taken up by non?parenchymal hepatic cells. This occurred because the enzyme preparations did not harbour the appropriate glycoprotein targeting sequences for uptake by membrane receptors leading to delivery to the nascent organelle.
Mannosephosphate Gaucher disease pathway residues are typically found on nascent lysosomal membranes, and are components of the pathway for endogenous and exogenous delivery of a large class of lysosomal proteins. This pathway is not in fact the relevant pathway for delivery of nascent glucocerebrosidase, which is a lysosomal membrane protein. Later, with the discovery of the macrophage mannose receptor, deglycosylation of purified preparations of human tissue glucocerebrosidase to reveal terminal mannose residues was successful in facilitating uptake of the enzyme molecules by enriched populations of macrophages.

Mannosylated human glucocerebrosidase, like other high?mannose glycoproteins, is taken up by a saturable process by macrophages. It presumably enters the phagolysosome compartment, where it may encounter the storage material that accumulates in the pathological macrophage that is the Gaucher’s cell. ( Detail from oxfordjournals.org on Gaucher disease pathway ).

Gaucher disease type 1

. A definitive diagnosis is made with genetic testing. As there are numerous different mutations, sequencing of the beta-glucosidase gene is sometimes necessary to confirm the diagnosis. Prenatal diagnosis is available, and is useful when there is a known genetic risk factor.
A diagnosis Gaucher disease type 1 can also be implied by biochemical abnormalities such as high alkaline phosphatase, angiotensin-converting enzyme (ACE) and immunoglobulin levels, or by cell analysis showing “crinkled paper” cytoplasm and glycolipid-laden macrophages.

Some lysosomal enzymes are elevated, including tartrate-resistant acid phosphatase, hexosaminidase, and a human chitinase, chitotriosidase. This latter enzyme has proved to be very useful for monitoring Gaucher’s disease activity in response to treatment, and may reflect the severity of the disease. ( Wikipedia diagnosis article on Gaucher disease type 1 )

Gaucher’s disease Type I effect

Gaucher’s disease Type I effect (or non-neuropathic type) is the most common form of the disease, occurring in approximately 1 in 50,000 live births. It occurs most often among persons of Ashkenazi Jewish heritage. Symptoms may begin early in life or in adulthood and include enlarged liver and grossly enlarged spleen (together hepatosplenomegaly); the spleen can rupture and cause additional complications.
Skeletal Gaucher’s disease Type I effect and bone disease may be extensive. Spleen enlargement and bone marrow replacement cause anemia, thrombocytopenia and leukopenia. The brain is not affected pathologically, but there may be lung and, rarely, kidney impairment.

Patients in this group usually bruise easily (due to low levels of platelets) and experience fatigue due to low numbers of red blood cells. Depending on disease onset and severity, type 1 patients may live well into adulthood. Many patients have a mild form of the disease or may not show any symptoms. ( Definition wikipedia about Gaucher’s disease Type I effect ).

Wednesday, June 6, 2012

Justin Bieber Style House at Los Angeles

Justin Bieber Style House at Los Angeles. The house features plenty of celebrity house styles amenities: glass chandeliers, retractable glass walls, gourmet kitchen, gym and massage room.
Justin Bieber Style House keeping with the California house style, the house features several outdoor spaces with enclosed gardens, terraces, and outdoor rooms.
The only thing holding Justin Bieber back from moving in immediately is the previous tenant. As reported earlier, the rumor is that Ashton Kutcher has been renting this Style House for $50,000 a month.
If the Justin Bieber take the Justin Bieber Style House with a classic mortgage, Justin Bieber can expect a monthly payment of $39,979, assuming a 20 % downpayment on a 30-year-fixed-rate mortgage.